SYMPTOMATIC ANIEMIAS INCLUDING SCHOOL ANIEMIA Symptomatic anemias deserve a separate discussion inasmuch as they depend so greatly upon their causes, and in many cases the recog nition of the etiology (hunger, for instance) is equivalent to a cure. The blood picture is a resultant of the injurious influences acting upon the blood-forming organs and the reactive capacity of these organs: the weaker these organs, the more readily will aniemia be produced.
Anaemia after cause of anfemia most easily recognized is hemorrhage (from an umbilical wound, in consequence of mehuna, infantile scurvy, epistaxis, hemorrhagic diathesis, intestinal haemorrhage, polypi, intussusception, gastric ulcer, intestinal parasites). If the acute losses of blood do not cause death, the infant will undergo rapid repair. Sometimes severe hLemorrhage is not followed by uninter rupted recovery, but may result in a prolonged and deleterious ancemia.
The therapy of acute anemia requires, after checking the rhage, hvpodermatic and intestinal saline infusions, heat, autotransfu sion by lowering the position of the head and wrapping up the extremi ties, and finally administering nourishing, but easily digestible food. Iron therapy is not very important in acute cases of loss of blood, be cause the latter in itself incites the blood-forming organs to renewed activity, and the ordinary food should as a rule contain sufficient iron. Infants, however, should receive small doses of iron because milk is deficient in iron. Suitable preparations are ferrum oxyd. solid). (P. G.) 0.03-0.05 Gm. (1-1 grain) three times daily, ferrum lactic. (P. G.) 0.01 Gm. grain) three times daily, liqu. ferri albuminat. (P. G.) S-10 drops three times daily: it is important, however, that the organs of digestion be in good condition.